![]() North Carolina powerboats for sale by owner. Michigan Wolverines football team. The 2. 01. 0 Michigan Wolverines football team represented the University of Michigan in the 2. NCAA Division I FBS football season. Michigan played its home games at Michigan Stadium in Ann Arbor, Michigan and competed in the Big Ten Conference. The season was the first since the renovation of Michigan Stadium, begun in 2. The Wolverines were led by third- year head coach Rich Rodriguez. After its week 1. Illinois, Michigan became bowl eligible for the first time since 2. ![]() Gator Bowl against Mississippi State on January 1, 2. The Wolverines finished the season sixth out of 1. Division I FBS teams in total offense, 1. ![]() Tickets for Concerts, Sports, Theatre and More Online at TicketsInventory.com. Michigan finished with an overall record of 7. After the season, head coach Rich Rodriguez and his staff were dismissed on January 5, 2. Several individual players had standout performances. Quarterback. Denard Robinson set several school records, became the first quarterback in college football history to rush and pass for 1,5. Heisman Trophy voting. He surpassed Drew Brees' single- season Big Ten Conference total offense record and the FBS single- season quarterback rushing record. He also won the 2. ![]() Great Lakes Fishing Boats for Sale. Sell your boat fast at www.walleye.com. Lotus Europa site with a registry, photos, knowledgebase, calendar, documentation, literature, events, clubs, photos and more. Here are some tips and tricks for troubleshooting and fixing laptop video problems. Video issues are very common within portable computers and with the following tips. Big Ten rushing title, the Chicago Tribune Silver Football for being the Big Ten's Most Valuable Player, and was named the Big Ten's Offensive Player of the Year. Jonas Mouton was the Big Ten's statistical champion in tackles, while Jordan Kovacs finished second. In conference games alone, Roy Roundtree led the conference in receiving yards per game, in part on the strength of a school single- game record 2. Illinois. Reserve quarterback Tate Forcier also set a school single- game completion percentage record with a 1. Bowling Green. The defense also set several records, including most points allowed and yards allowed. Preseason. Harding H. S. 6 ft 6 in (1. 9. Jan 1. 7, 2. 01. 0 Scout: Rivals: 2. Sports: N/A ESPN grade: 7. Jake Ryan. LBWestlake, Ohio. St. Ignatius H. S.
Jan 1. 8, 2. 01. 0 Scout: Rivals: 2. Sports: N/A ESPN grade: 7. Terrence Talbott. DBHuber Heights, Ohio. Wayne H. S. 5 ft 9. Aug 1. 5, 2. 00. 9 Scout: Rivals: 2. Sports: N/A ESPN grade: 7. Terry Talbott. DTHuber Heights, Ohio. Wayne H. S. 6 ft 3. Aug 1. 5, 2. 00. 9 Scout: Rivals: 2. Sports: N/A ESPN grade: 7. Ray Vinopal. DBYoungstown, Ohio. Cardinal Mooney H. S. 5 ft 1. 0. 5 in (1. Dec 4, 2. 00. 9 Scout: Rivals: 2. Sports: N/A ESPN grade: 6. Austin White. RBLivonia, Michigan. Stevenson H. S. 6 ft 0 in (1. Jul 1. 5, 2. 00. 9 Scout: Rivals: 2. Sports: N/A ESPN grade: 7. Ken Wilkins. DEWashington, Pennsylvania. Trinity H. S. 6 ft 3 in (1. Jun 9, 2. 00. 9 Scout: Rivals: 2. Sports: N/A ESPN grade: 7. D. J. Williamson. WRWarren, Ohio. Warren G. Harding H. S. 6 ft 1. Mar 1. 4, 2. 00. 9 Scout: Rivals: 2. Sports: N/A ESPN grade: 7. Overall recruiting rankings: Scout: 1. Rivals: 2. 0 ESPN: 1. Note: In many cases, Scout, Rivals, 2. Sports, and ESPN may conflict in their listings of height and weight. In these cases, the average was taken. ESPN grades are on a 1. Sources: Preseason award watch lists. Iowa. Michigan Stadium . Wisconsin. Michigan Stadium . Ohio State. Ohio Stadium . Mississippi State*Ever. Bank Field . All times are in Eastern Time. Game summaries. Michigan started the second quarter with another touchdown, this time with a 3- yard run by Michael Shaw. All of Connecticut's scoring came in the second quarter: a 3. Dave Teggert, and then a 2- yard run by Jordan Todman. The only scoring of the third quarter came on a 2. Michigan field goal by Brendan Gibbons. Connecticut managed to enter Michigan's red zone during the third quarter, but D. J. Shoemate fumbled the ball after a hit by J. T. Floyd and Obi Ezeh recovered it to prevent Connecticut from scoring. In the fourth quarter, Smith caught an 1. Robinson; however, the extra point attempt was no good. Robinson had 2. 9 carries and completed 1. Michigan's 4. 73 total yards. Michigan committed no turnovers, the first time since the game against Miami (Ohio) on September 6, 2. With the win, the Wolverines improved to 1. Michigan won the previous year's contest on a Tate Forcier to Greg Mathews touchdown pass with 1. Michigan responded with three unanswered touchdowns. First was a 3. 1- yard reception by Roy Roundtree midway through the first quarter, then a 1- yard run by Stephen Hopkins near the end of the first, and later an 8. QB Denard Robinson, the longest in Notre Dame Stadium history. After halftime, the Fighting Irish responded with two touchdowns and a field goal. First was a 5. 3- yard pass to TJ Jones, followed by a 2. David Ruffer. Next was a 9. Kyle Rudolph, which put Notre Dame ahead late in the game. Michigan retook the lead with 2. Denard Robinson. Michigan's defense held off Notre Dame on its final drive of the game, with Crist's final pass sailing over everyone and out of the end zone. The rushing yards set a new high for yards on the ground in one game by a Big Ten quarterback, as Robinson broke the league record of 2. Mike Kafka at Minnesota on November 1, 2. Robinson became the ninth quarterback in NCAA history to rush and pass for 2. West Virginia's Pat White against Pittsburgh on November 1. The last 2. 00- yard rusher for Michigan was Mike Hart, who had 2. Eastern Michigan on October 6, 2. On the opening drive, UMass drove to the Michigan 1. On Michigan's first possession, Denard Robinson threw an interception on the second play (and first pass) of the drive. After a defensive stop, Michigan drove 9. Martavious Odoms and 4. Kelvin Grady. Michael Shaw scored the touchdown on a 1- yard run. Thirty- five seconds into the second quarter, UMass retook the lead on a 7- yard touchdown run by Jonathan Hernandez, capping a 6. Midway through the second quarter, Michigan drove 6. Robinson), but Seth Broekhuizen missed a 3. In the closing minutes of the first half, UMass drove 7. Hernandez. On Michigan's first play from scrimmage after the UMass touchdown, Robinson threw a 6. Darryl Stonum with 1: 0. On the second play from scrimmage after the kickoff, Jordan Kovacs forced a fumble by Hernandez, and Michigan recovered the ball at their own 4. Michigan quickly drove 5. Robinson to Stonum with 1. On their second possession of the second half, Michigan drove 9. Robinson. The score marked Michigan's fourth touchdown in four drives as Michigan scored 2. UMass took the 1. UMass scored on a 7- yard touchdown run by Havens at 1. On Michigan's first possession of the fourth quarter, Shaw led the way with a 5. Michigan ahead 4. UMass responded with its second touchdown of the quarter on a five- yard pass from Havens to Julian Talley, but missed on the two- point conversion attempt. After a three- and- out by Michigan, UMass blocked Michigan's punt and drove for its third fourth- quarter touchdown aided by a pass interference penalty against Michigan's James Rogers. With 2: 0. 5 left to play, and Michigan leading 4. This was the first meeting between the schools since 2. Michigan emerge victorious 4. On its second possession, Michigan drove the ball 8. Michigan began its third possession at its own 9- yard line. Robinson ran 4. 6 yards on the first play of the drive, but he injured his knee and did not return to the game. Devin Gardner substituted for Robinson and concluded the drive with an 1. Jeremy Gallon. With Michigan's lead cut to 7 points, Tate Forcier entered the game late in the second quarter. Forcier completed all 6 of his passes in leading the team on 6. Michigan a 2. 8. After the safety, Forcier completed a 2. Roy Roundtree, which led to a 2- yard pass from Forcier to John Mc. Colgan. On Bowling Green's next drive, Jonas Mouton intercepted a pass at the Bowling Green 4. After a 1. 4- yard pass from Forcier to Gallon, Vincent Smith scored on an 8- yard run to give Michigan a 4. Bowling Green then drove 6. Jordan Hopgood. Michigan held Bowling Green scoreless in the fourth quarter and added three more touchdowns on runs of 1 yard by Vincent Smith, 5 yards by Fitzgerald Toussaint and 7 yards by Devin Gardner. Toussaint also had a 6. It is the most recorded by Michigan against an FBS program, surpassing the 7. Mississippi in the 1. Gator Bowl. Michigan's three quarterbacks combined to complete 2. This marked the first time Michigan had won its first four games in back- to- back seasons since the 1. During the previous meeting between the schools, Michigan won the game on a controversial interception call which saw Donovan Warren intercept Indiana quarterback Ben Chappell on what turned out to be Indiana's final play of the game. Michigan responded less than a minute later on its second play from scrimmage with a 7. Denard Robinson. On its second possession, Robinson led an 8. Roy Roundtree. On Michigan's third possession of the first quarter, Robinson completed a 7. Roundtree who was tackled at the 1- yard line. However, Robinson fumbled the snap and the ball was recovered by Indiana. Following the fumble, Chappell led Indiana on a five- minute, 9. Darius Willis. Michigan re- took the lead 2. The half ended with a 7. Indiana and a 3- yard touchdown pass from Chappell to Damarlo Belcher as time ran out. Demarlo Willis tied the game at 2. Michigan once again took the lead less than a minute later with a 5. Vincent Smith. The score remained 3. Late in the fourth quarter, Chappell led Indiana on a six- minute, 8. On fourth down with 1: 2. Chappell completed a 1. Darius Willis to tie the game at 3. The Wolverines got the ball back at their own 2. Robinson carried the ball three times to take the ball to the Indiana 4. Hemingway who was tackled at the Indiana 4- yard line. Robinson ran the ball into the endzone for the winning touchdown with 1. It was Michigan's 1. Indiana, dating back to 1. The victory also gave Michigan its first 5. He contributed 4. Michigan history. With 2. 17 rushing yards and 2. Robinson became the first player in Division I FBS history to have two regular season games with both 2. Michigan's defense gave up 5. Michigan football team. Indiana's total of 4. Michigan opponent. Indiana quarterback Ben Chappell passed for 4. Congenital disorders of the optic nerve: excavations and hypoplasia. Top of page. Excavations of the optic disc. Optic disc coloboma. Definition Optic disc coloboma (Figure 1) comprises a clearly demarcated bowl- shaped excavation of the optic disc, which is typically decentred and deeper inferiorly. Aetiology Coloboma of the optic disc is thought to result from abnormal fusion of the two sides of the proximal end of the optic cup. The condition can occur in association with multiple congenital abnormalities indicative of an insult to the developing foetus during the sixth week of gestation. Optic nerve coloboma may occur sporadically or be inherited with an autosomal dominant inheritance. It has recently been shown to be associated with PAX2 gene mutations as part of the renal- coloboma syndrome. Expression of PAX2 is restricted to cells of astrocytic lineage both during retinal development and in adulthood. Using immunohistochemistry, it has been found that adult retinal cells with the antigenic phenotype present in mature perinatal astrocytes are found only in the region surrounding the optic nerve head, and that astrocyte precursor cells expressing PAX2 are found in a small region surrounding the optic nerve during early development. It has been argued that these findings suggest that coloboma formation may be associated with impaired astrocyte differentiation during development. The association of optic nerve coloboma with dysgenesis of the internal carotid artery and transphenoidal encephalocele with hypopituitarism has led to the suggestion that the link between these malformations is abnormal neural crest cell development. Clinical features Unilateral and bilateral optic disc colobomata occur with similar frequencies. The coloboma occupies the lower part of the optic nerve head. The neuro- retinal rim is absent inferiorly but is usually identifiable superiorly. In cases in which the adjacent inferior retina and choroid are deficient, microphthalmia may also be evident. Visual acuity is reduced to varying degrees. Careful analysis of the photographic appearances of colobomata involving the optic nerve has shown that the only feature that relates to visual outcome is the degree of foveal involvement by the coloboma. The size of the coloboma, the colour of the optic nerve, and the presence of subfoveal pigment change are not related to visual outcome. Significant refractive error and anisometropia are common. Progressive optic nerve cupping and neural rim decrease have been documented in a patient with bilateral autosomal dominant optic nerve colobomas with no evidence of raised intraocular pressure and remarkably, no progressive visual field loss. Circumferential intrascleral smooth muscle has been observed histologically. Complications A small proportion of cases are associated with cysts arising from the optic nerve sheath, which communicate with the subarachnoid space. Rarely such cysts can enlarge and lead to compressive optic neuropathy. Peripapillary choroidal neovascularisation has been described in association with optic nerve coloboma. Retinal detachment is also a recognised complication, and remarkably, spontaneous re- attachment may occur. The source of the subretinal fluid is not known but could derive from fluid entering the retrobulbar space from surrounding orbital tissue, or from the choriocapillaris, or from CSF. In contrast to retinochoroidal colobomas, rhegmatogenous detachment is probably not a recognised association. Contrary to what is commonly taught, basal encephalocele is a rare association with optic nerve coloboma. Associations There is a wide range of associations. These have been reviewed by Brodsky. CHARGE association (coloboma, choanal atresia, congenital heart disease, and multiple other abnormalities), Walker–Warburg syndrome, Goltz focal dermal hypoplasia, Aicardi syndrome, Goldenhar syndrome, and linear sebaceous naevus syndrome. More recently, associations with Dandy Walker malformation. PAX2 transcription)4, 5, 6, 7 have also been described. Treatment A trial of patching may result in improvement of vision in the child presenting early in life, and optimal refractive correction may be indicated. Morning glory anomaly. Definition The morning glory optic disc (Figure 2) anomaly is a congenital optic disc dysplasia in which a conical excavation of the posterior fundus includes the optic disc and is filled with glial tissue. The term reflects the morphological similarity to the flower of the morning glory plant. Aetiology Morning glory syndrome is ostensibly a sporadic condition but it has been described occurring in isolation in a parent and child. The pathogenesis of the condition is unknown. One hypothesis argues that the condition results from failure of closure of the foetal fissure and that it is a variant of optic nerve coloboma. Alternatively, a primary mesenchymal abnormality has been postulated on the basis of the glial tuft, the scleral and vascular abnormalities, and the finding of adipose and smooth muscle tissue around the terminal optic nerve. An alternative argument is that the symmetrical excavation of the optic nerve head, which characterises the disorder, is suggestive of dilation due to dysgenesis of the terminal optic stalk, which fails to close, leading to persistent excavation of the optic nerve head. The central gliosis and the vascular pattern suggest primary neuro- ectodermal dysgenesis. Morning glory syndrome is more common in female population and (in the USA) occurs less commonly in black people. Clinical features Morning glory syndrome is usually unilateral and is characterised by a funnel- shaped, excavated optic disc (which may even include the macula) with central glial tissue surrounded by chorioretinal pigmentary disturbance. The central glial material may be either elevated or recessed. The retinal vessels are increased in number and appear to arise from the disc margin from where they tend to run to the peripheral retina in a straighter course than usual. Fluorescein angiography has revealed arteriovenous communications near the optic disc. The visual acuity is usually reduced, being in the region of count fingers to 6/6. Rarely the acuity can be as good as 6/6. The remarkable phenomenon of contractile movements of the optic disc has been reported on a number of occasions. In one reported case, phasic contraction was observed by scanning laser ophthalmoscopy over 2- s periods of contraction and 2. The contractile movement was not related to exposure to strong light or the Valsalva manoeuvre but it seemed to be induced by massage of the eyeball. In another reported case, both the contractions and the periods of relaxation lasted for 4- s, occurring between three and five times a minute. Histologically, a cuff of smooth muscle tissue has been identified within the terminal optic nerve and this may account for the observed movement. Associations and complications Morning glory syndrome tends to be an isolated condition and does not occur as part of a multisystem disorder. It can however be associated with transphenoidal encephalocele and hypopituitarism. Patients with this condition tend to have a wide head and hypertelorism associated with a flattened nasal bridge. The encephalocele bulges into the nasopharynx where it can obstruct respiration. Incorrect identification and management of this pathology as a nasal polyp has been reported to lead to a fatal outcome. Rhegmatogenous retinal detachment (which can now be successfully treated) can be associated with a break at the optic disc margin. Remarkably, optic nerve fenestration has been reported to lead to resolution of serous retinal detachment, suggesting that the source of subretinal fluid can be CSF. Spontaneous resolution of retinal detachment has also been reported. Subretinal neovascularisation both under the peripapillary retina. Treatment When the condition is first seen in young children, a trial of occlusion is warranted but should not be continued if improvement in vision does not take place. Peripapillary staphyloma. Definition Peripapillary staphyloma consists of a deep excavation of the area of the fundus surrounding the optic disc (Figure 3). Epidemiology The condition is sporadic, very rare, usually unilateral, and is not inherited. Aetiology The aetiology is unknown. It appears to arise as a sequel to disturbance of scleral development at about 2. Clinical features The optic nerve may look normal or manifest temporal pallor but it is situated within an excavated defect of the sclera lined by retina and choroid, which tend to be atrophic in appearance. Like optic nerve coloboma and morning glory syndrome, spontaneous contractility and relaxation of the staphyloma has been observed. Vision is usually markedly reduced in the affected eye but has occasionally been reported as near normal. A centrocaecal scotoma may be present in eyes with reduced visual acuity. Myopia or emmetropia most commonly accompanies the condition. Associations In most reported cases, peripapillary staphyloma occurs in isolation and is unilateral. Two out of six patients with frontonasal dysplasia in association with basal encephalocele were found to have either unilateral or bilateral peripapillary staphyloma. Both facial capillary haemangioma. Optic nerve hypoplasia. Definition Optic nerve hypoplasia (ONH) is seen ophthalmoscopically as an abnormally small optic nerve head. Recently, the definition has been extended to include cases of optic nerve head cupping, which accompany periventricular leucomalacia (PVL). In both conditions, there are fewer fibres in the optic nerve from the time of birth. Epidemiology ONH is among the three leading causes of blindness in children in the United states. The other causes are cerebral damage and retinopathy of prematurity. Aetiology In most cases, ONH is a sporadic occurrence for which there is no identifiable cause. There are, however, a number of associations, which are listed in Table 1. Bertram powerboats for sale by owner.
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